Fri, Jul-06-07, 14:59
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Senior Member
Posts: 1,162
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Plan: Atkins
Stats: 228/195/188
BF:
Progress: 83%
Location: Erie PA
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More about Fructose
http://www.medscape.com/viewarticle/559344?src=mp
Quote:
Relative to baseline, 24-hour postprandial triglyceride profiles were increased by 212% ± 59% in the fructose-consuming group (P < .0001). In contrast, levels declined by about one third (−30% ± 23%) in the glucose-consuming group. In addition, fasting plasma levels of low-density lipoprotein cholesterol (LDL-C); apolipoprotein B; and small, dense LDL-C, as well as postprandial levels of remnant lipoprotein (RLP)-triglycerides and RLP-cholesterol, were all significantly increased (P < .01) in the fructose group. By comparison, these levels remained unchanged in the glucose group.
Fructose-consuming participants also demonstrated increased plasma concentrations of the atherogenic risk factors oxidized LDL-C (P < .0001) and intracellular adhesion molecule (P < .05), but those consuming glucose did not.
"Consumption of sugar-sweetened beverages containing fructose has increased by 135% from 1977 to 2001 and may be a contributing factor to an increased incidence of metabolic syndrome," the authors note in their abstract.
According to Dr. Havel, most soft drinks in the United States are sweetened with high-fructose corn syrup, which is a mixture of about 55% fructose and 45% glucose.
"It is known that fructose, after being metabolized by the liver, is more likely to go into a lipogenic pathway than glucose," Dr. Havel noted. "So these results were not surprising to us, but the magnitude of some of the changes was striking," he added.
"While this is an interim report, the findings do suggest that persons at risk for cardiovascular disease, diabetes, or hyperlipidemia should limit consumption of fructose-sweetened beverages. It is unclear, however, whether a nonatherogenic level of fructose consumption exists, and what that might be."
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