I just wanted to say a few things.
It is simply scientifically incorrect to believe that individual biological make up has no role in how well our metabolism can handle sugar. Kent, I am sorry, but science is against you when you claim that all people have the exact same metabolic plasticity and tolerance for carbohydrate from birth. Some people get sick on higher carb, some people won't.
Phenotypes which were produced in environments where the people thrived on very low sugar, high fat and protein diets tend to develop IRS more readily than phenotypes which are the product of grain dependant societies. Observe the native american, or african american population (both peoples who were produced by cultures in which the diet is traditionally high-fat and low-sugar) in contrast to asian americans and even european americans (where moderate in fat and a little higher in sugar diets were more common). Despite eating roughly the same carb-heavy diet as asian americans and european americans, the rates of obesity, heart disease and diabetes in native americans and african americans are much, much higher.
While the obvious answer seems to be individual genetically determined metabolic variance, I doubt my saying this would convince you. Perhaps some hard evidence is in order. Scientists have discovered genetic markers for developing IRS (insulin resistance syndrome), or what could also be called having a poor sugar metabolism.
http://diabetes.diabetesjournals.or...t/full/51/3/841
Quote:
Insulin resistance syndrome (IRS)-related phenotypes, such as hyperinsulinemia, obesity-related traits, impaired glucose tolerance, dyslipidemia, and hypertension, tend to cluster into factors. We attempted to identify loci influencing the factors of IRS-related phenotypes using phenotypic data from 261 nondiabetic subjects distributed across 27 low-income Mexican-American extended families. Principal component factor analyses were performed using eight IRS-related phenotypes: fasting glucose (FG), fasting specific insulin (FSI), BMI, systolic blood pressure (SBP), diastolic blood pressure (DBP), HDL cholesterol, ln triglycerides (ln TGs), and leptin (LEP). The factor analysis yielded three factors: factor 1 (BMI, LEP, and FSI), factor 2 (DBP and SBP), and factor 3 (HDL and ln TG). We conducted multipoint variance components linkage analyses on these factors with the program SOLAR using a 10–15 cM map. We found significant evidence for linkage of factor 1 to two regions on chromosome 6 near markers D6S403 (logarithm of odds [LOD] = 4.2) and D6S264 (LOD = 4.9). We also found strong evidence for linkage of factor 3 to a genetic location on chromosome 7 between markers D7S479 and D7S471 (LOD = 3.2). In conclusion, we found substantial evidence for susceptibility loci on chromosomes 6 and 7 that appear to influence the factors representing the IRS-related phenotypes in Mexican-Americans.
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Other studies have drawn similar conclusions: Due to individual biological make up, some people quite simply lack metabolic plasticity to eat a high-sugar diet and thrive on it. Though most studies have been confined to recently-modernized peoples freshly coming off of a high-fat, low-sugar diet like NAs and AAs, the evidence is strong that we don't all have the same genetically determined dietary plasticity. There is no reason to believe IRS phenotypes don't exist within european american population, or even the asian american population.
While I think it is safe to say that evidence is strong enough to smash the theory that a higher carbohydrate makes
all people sick, I don't think it is logically consistent to extrapolate from this that a higher carbohydrate diet is
optimal for those who don't get sick on it. In other words, "carb types" may not exist at all, they just simply have more options because they can "tolerate" sugar more effectively, so to speak. Absence of disease is not the same thing as thriving, and those who don't get sick on high carb may actually do better on lower-carb.
However, I've yet to see evidence either way, but I tend to fall towards the metabolic typing theory in that, yes, some people not only tolerate carbs better but they are better served by higher carb diets. I do think it is quite reasonable to assume that those with good carbohydrate metabolism would do best on a higher-carbohydrate diet than those with a poor carbohydrate metabolism. Even if they see no benefits when they eat more carbs and they are simply capable of "tolerating it", the increased dietary variety and decrease in dietary restrictions have to have some psychological benefit. Stress and its hormonal effects have long been shown to have deleterious physiological side effects.
Plus, I am sure being able to eat more carbs has a nutritional benefit as well. If you can eat more fruit and veggies and grain, you should, as they are excellent sources of minerals and phytochemicals.
Another thing I wanted to say is that it is incorrect to extrapolate that an all meat diet is healthy, because the traditional inuit diet is virtually all meat and they enjoy good health. First of all, the types of meat the inuit eat are natural, free range animals and aquatic life - these fats and meats are replete with vitamins, anti-oxidants, fatty acids, and minerals not found in grain-fed western meat. Second of all, it is very likely that the inuit have a very different biological make up than western man. The traditional native american diet was for the most part very low in sugar. While it has been established that native americans for the most part lack metabolic plasticity to handle a high sugar diet (as they were never exposed to it before), what is less clear is whether or not they have a
more sophisticated fat metabolism.
In other words, the metabolic typing theory might have some validity in that some people can handle high-fat better than others. I have no idea whether or not this is true, more research is needed.