Mon, Jan-11-16, 10:50
|
 |
Senior Member
Posts: 4,068
|
|
Plan: Very LC, Higher Protein
Stats: 227/186/185
BF:
Progress: 98%
Location: Herndon, VA
|
|
Quote:
|
Originally Posted by nwlifter
In a few of those studies I posted they did trace it. Of course, even indirectly, all substrates come from diet, but the key FFA they have traced, and traced it to organ cell disfunction is palmitate. The excess comes from 2 places. Visceral fat, and creation from dietary carbs, namely sugar. FFA creation needs glucose to donate the glycerol molecule as the 'backbone' of the FFA. If glucose is low in the blood, trig production is crippled. But also, palmitate creation is very limited.
|
That's the key. Dietary consumption of a variant does not mean that the variant in question is introduced directly by consumption of that variant. This is the same mistake made when Keys purported that consumption of fatty foods resulted in dangerous blood cholesterol levels. Unfortunately, this is still a strong perception today, and it likely will take years for the medical community and the government to move away from this thinking. It's not intuitive to believe that FFA substances like palmitate can come from dietary carbs and not dietary fats; however, that's exactly what's happening.
Good discussion and one that many on this forum are very familiar with and plugged in to. Explore the other forum topics, as I'm sure you'll find a lot of good information consistent with your research and current beliefs. There are lots of knowledgeable posters here who contribute regularly.
|