"The 10-year cohort study by Jee et al collected the health data of 1,298,385 Koreans (829,770 men and 468,615 women), aged between 30 and 95 years, found the risks for
various cancers were positively related to the individual’s fast serum glucose level and a diagnosis of diabetes mellitus, but did not find a notable link to body mass index. In other words, being slim alone does not reduce the risks for cancers. Although the study covered variable cancers, the colorectal and liver cancers in men were statistically significant in related to the fasting serum glucose (0.03 each) only next to the esophageal (0.007) pancreatic (0.009) cancers. At the same time, the colorectal cancer in women also had a significant trend with the fasting serum glucose level. While the fasting blood serum glucose level is positively linked to the risk of cancers including colorectal cancer, those study participants in the control group, whose fasting serum glucose was below 90 mg%, still yielded a substantial number of incidences. This indicates positive association of the serum glucose level with the risks of cancers might be beyond that of the fasting.
Despite several studies denied the connection between the incidence of colorectal cancer and glycemic index, glycemic load, and carbohydrates consumption, [7, 8] the data from nearly 1.3 million individuals used in Jee’s study can hardly be ignored.
In addition, more recent studies supported the link between the risk of colorectal cancer and the consumption of carbohydrates. [9, 10] Gnagnarella P et al summarized, “This comprehensive meta-analysis of GI and GL and cancer risk suggested an overall direct association with colorectal and endometrial cancer.” [11] Barclay AW et al concluded,
“The findings support the hypothesis that higher postprandial glycemia is a universal mechanism for disease progression.” [12]
Several studies have found postprandial hyperglycemia triggers inflammation. [13] A review article by Coussens LM and Werb Z cites, “It is now evident that inflammatory cells have powerful effects on tumour development. Early in the neoplastic process, these cells are powerful tumour promoters, producing an attractive environment for tumour growth, facilitating genomic instability and promoting angiogenesis”, and, “It is clear that anti-inflammatory therapy is efficacious towards early neoplastic progression and malignant conversion. In a fully developed malignancy, there are ‘excess’ inflammatory cells in the tumour microenvironment.” [14] The latter substantiates the observation that the use of Aspirin reduces the deaths from colon cancer.” [15]"
http://www.carbohydratescankill.com...es-colon-cancer
So the Dr. Oz diet will make you thin AND give you butt cancer