The context here is single dose. Can't extend any conclusion to longer term. For example, if they found that glycogen was retained, it's because of that effect I'm talking about. But then, that same effect also causes insulin to drop, which in turn allows more glycogen to be released, later in subsequent doses if done consistently, which they didn't try.
As for the bonk, it's all wrong. It's thought that we run out of glycogen, when in fact we run out of ketones (and possibly of fatty acids). Remember, it's all in the context of carbing up precisely to avoid a bonk. Well, that causes insulin to rise with every dose of carbs, often throughout a race. Each time carbs go in, insulin goes up, ketogenesis is inhibited, ketones eventually go to zero, bonk. BG can't go to zero, hypo is impossible - carbs coming in, see? Can't be because there's not enough insulin to push glucose into cells - there's hyperinsulinemia from all the carbs coming in, see? Maybe it's a form of insulin resistance, but then why does ketogenesis drop to zero and ketones drop to zero, which can only occur when the liver responds - is sensitive - to the hyperinsulinemia?
It's also possible there's something else going on. For example, we use certain drugs to manipulate cellular respiration and oxidation. If this is possible, it's because the pathways are there. Maybe it's the fight-flight response where some hormones take over, a sort of nitro boost. Glucose isn't it, it's something else, something more powerful and quicker than just oxygen. But if we disrupt normal fuel supply in such a way, we could trigger this nitro boost. At face value, we could conclude it's the glucose, but that's just the trigger for a sequence of events that leads to nitro boost.
Another thing, this:
Quote:
"Often these supplements are marketed as a means of improving athletic performance but in this case, the research tells a very different story," says Little. "On top of that, the long-term impacts of artificially increasing blood ketone levels -- essentially tricking the body into thinking it is in a state of starvation -- is completely unknown."
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If they believe that's what's going on, they gotta do their homework. If the body is in a state of starvation, how can this possibly be done by eating food, i.e. a ketogenic diet? And how is it possible that starvation is anything bad, when everything runs normally to supply the body with all it needs, by releasing stored fat and stored glycogen and providing ample fuel in the form of ketones? Low-carb allows this to continue, no disruption. High-carb completely disrupts this and triggers other things down the line. Stress hormones? Check.
Sorry, didn't mean to sound contentious. Just saying it's a single dose experiment, can't conclude anything long-term from it.