Mon, May-31-10, 02:17
|
 |
Registered Member
Posts: 2,886
|
|
Plan: Dr Dahlqvist's
Stats: 205/152/160
BF:
Progress: 118%
|
|
New blog by Nephropal section on Coconut oil below
the blog starts with a bit on Cholesterol and the wisdom? of reducing levels.
I haven't commented yet but make no mistake people with the lowest cholesterol levels live shorter livess and one of the reason why people with the lowest cholesterol level die soonest is Vitamin D.
The lower your skin's cholesterol level the less vitamin D it is capable of making.
Your immune function is based on the vitamin D response to pathogens.
Low vitamin D inevitably means immune response is compromised. But this thread is about Coconut oil and Nephropal explains why it may be better to go form the VCO then the cold pressed.
Quote:
Now to my favorite fat - coconut oil which is 94% saturated fat (lauric acid). A recent Brazilian study showed that coconut oil INCREASED HDL and LOWERED LDL: HDL ratio in obese females (abstract is the second one below).
A Chinese study (click) has revealed that the coconut oil extract monolaurin has anti-microbial effects against E. coli and Bacillus subtilis but not Staphylococcus aureus.
A recently study from India showed that virgin coconut oil may be superior to dry press.
abstract:
Can J Physiol Pharmacol. 2009 Aug;87(8):610-6.
Wet and dry extraction of coconut oil: impact on lipid metabolic and antioxidant status in cholesterol coadministered rats.
Nevin KG, Rajamohan T.
Department of Biochemistry, University of Kerala, Kariavattom, Thiruvananthapuram, Kerala 695 581, India.
Because coconut oil extracted by wet process (virgin coconut oil, VCO) is gaining popularity among consumers, this study was conducted to evaluate VCO compared with coconut oil extracted by dry process (copra oil, CO) for their influence on lipid parameters, lipid peroxidation, and antioxidant status in rats coadministered with cholesterol. VCO, CO, and cholesterol were fed in a semi-synthetic diet to 24 male Sprague-Dawley rats for 45 days. After the experimental period, lipid and lipid peroxide levels and antioxidant enzyme activities were observed. Chemical composition and antioxidant properties of the polyphenolic fraction from VCO and CO were also analyzed. The results showed that lipid and lipid peroxide levels were lower in VCO-fed animals than in animals fed either CO or cholesterol alone. Antioxidant enzyme activities in VCO-fed animals were comparable with those in control animals. Although the fatty acid profiles of both oils were similar, a significantly higher level of unsaponifiable components was observed in VCO. Polyphenols from VCO also showed significant radical-scavenging activity compared with those from CO. This study clearly indicates the potential benefits of VCO over CO in maintaining lipid metabolism and antioxidant status. These effects may be attributed in part to the presence of biologically active minor unsaponifiable components.
____________________________________________________
Brazilian abstract
Lipids. 2009 Jul;44(7):593-601. Epub 2009 May 13.
Effects of dietary coconut oil on the biochemical and anthropometric profiles of women presenting abdominal obesity.
Assunção ML, Ferreira HS, dos Santos AF, Cabral CR Jr, Florêncio TM.
Faculdade de Nutrição, Universidade Federal de Alagoas, Maceió, AL 57072-970, Brazil.
Abstract
The effects of dietary supplementation with coconut oil on the biochemical and anthropometric profiles of women presenting waist circumferences (WC) greater than 88 cm (abdominal obesity) were investigated. The randomised, double-blind, clinical trial involved 40 women aged 20-40 years. Groups received daily dietary supplements comprising 30 mL of either soy bean oil (group S; n = 20) or coconut oil (group C; n = 20) over a 12-week period, during which all subjects were instructed to follow a balanced hypocaloric diet and to walk for 50 min per day. Data were collected 1 week before (T1) and 1 week after (T2) dietary intervention. Energy intake and amount of carbohydrate ingested by both groups diminished over the trial, whereas the consumption of protein and fibre increased and lipid ingestion remained unchanged. At T1 there were no differences in biochemical or anthropometric characteristics between the groups, whereas at T2 group C presented a higher level of HDL (48.7 +/- 2.4 vs. 45.00 +/- 5.6; P = 0.01) and a lower LDL:HDL ratio (2.41 +/- 0.8 vs. 3.1 +/- 0.8; P = 0.04). Reductions in BMI were observed in both groups at T2 (P less than p =" 0.005). Group S presented an increase (P less than 0.05) in total cholesterol, LDL,and LDL:HDL ratio, whilst HDL diminished (P = 0.03). Such alterations were not observed in group C. It appears that dietetic supplementation with coconut oil does not cause dyslipidemia and seems to promote a reduction in abdominal obesity.
___________________________________________
In an additional study (on rabbits) from India, they "conclude that in the absence of cholesterol supplementation, coconut oil intake up to 30% of daily energy supply did not cause hypercholesterolemia or oxidative stress in rabbits. " Oxidation of lipids was also lower in the coconut oil group. Meaning the coconut oil was less prone to oxidation by free radicals. This could be due to coconut oil's Vitamin E content. (click here for the full article). In the article, in the discussion section, the authors note that coconut oil has more anti-oxidant properties than olive oil and fish oil in animal studies. Why? Unlike saturated fat which has NO double bonds, polyunsaturated fat has two and monosaturated fat has one double bonds. These bonds are prone to disruption by free radicals. (click here my previous post on fats: introduction) Thus monounsaturated fats like avocados will be less prone to disruption than polyunsaturated fats -like sunflower oil. Thus coconut oil has better anti-oxidant capacity versus sunflower oil in this study. The abstract is below.
abstract
J Atheroscler Thromb. 2010 Feb 26;17(2):213-8. Epub 2009 Dec 24.
Effect of high fat diet without cholesterol supplementation on oxidative stress and lipid peroxidation in New Zealand white rabbits.
Sabitha P, Vasudevan DM, Kamath P.
Department of Biochemistry, Amrita School of Medicine, Kerala, India. sabitha.palazhy~gmail.com
AIM: Dietary fats may affect coronary artery disease risk by influencing factors other than serum cholesterol. The effect of diets containing coconut oil and sunflower oil without cholesterol supplementation on oxidative stress and lipid peroxidation were studied in male New Zealand White rabbits. METHODS: Animals assigned to four groups (control, cholesterol-fed, coconut oil-fed and sunflower oil-fed), given an isocaloric diet and studied for 6 months. The lipid profile, reduced glutathione, glutathione peroxidase, superoxide dismutase, vitamin C and lipid peroxidation were evaluated at the beginning of the study, at the third month and at the end of the study period. RESULTS: Serum lipid values did not show significant variation between animals fed coconut oil and sunflower oil, but total cholesterol, triglycerides and LDL-cholesterol were significantly higher and HDL-cholesterol was reduced in cholesterol-fed animals. Lipid peroxidation was higher in cholesterol-fed and sunflower oil-fed rabbits compared to controls and coconut oil-fed rabbits. Though other parameters such as reduced glutathione, glutathione peroxidase, superoxide dismutase and ascorbate did not vary between the two oil-fed rabbit groups, cholesterol-fed rabbits showed severe oxidative stress. CONCLUSION: We conclude that in the absence of cholesterol supplementation, coconut oil intake up to 30% of daily energy supply did not cause hypercholesterolemia or oxidative stress in rabbits.
POSTED BY DR. T AT 7:37 AM
|
|
|
Linear Mode
