Some people are quite unlucky when it comes to plaque's. We all have a genetic pre-disposition to the first stage of plaque (called a fatty streak) formation. Some have lots some don't. They can also form almost anywhere. The basic theory of how a plaque develops is that, due to damage of that fatty streak. From there various blood borne products are recruited. It is believed that a high amount of cholesterol leads to more cholesterol in the plaque making it more unstable quicker.

Now the problems that exist with this theory are the ones that are coming into play now. Cholesterol concentration is not the be all and end all. But.. HDL is a fantastic antioxidant, if we have more of it then the oxidative species in the blood don't damage other things they are mopped up by the HDL. (http://www.ncbi.nlm.nih.gov/pubmed/18241620). This is why we want lots off it. LDL is not a good antioxidant.

So to build up HDL levels, one cut down on oxidative stresses (carbohydrates, and incomplete carbohydrate synthesis) which you are doing :-). Exercise increases HDL in the body as mentioned (http://www.sciencedirect.com/scienc...2604958390152X), which again you are addressing.

As for the medication, you fit into one of the seriously unlucky groups where it is hard to manage, if your arteries are not that bad then it is only prophylactic help that can be provided.

The aspirin and clopidogrel are to stop platelet aggregation around and emboli.
The Statin's main important role in your case is stabilisation of any plaque's in your body.

The last two are anti-hypertensives, as control of your BP is now really important and they are probably aiming for 130 systolic.

Hope that is of some help :-)