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Tom Matthe
Wed, Jun-26-02, 20:56
Medscape Medical News Nuts May Lower Risk of Diabetes

Laurie Barclay, MD

Medscape Medical News 2002. =A9 2002 Medscape

June 19, 2002 -- New data presented June 16-17 at the 62nd
Scientific Ses= sions of=20 the American Diabetes Association
suggest that eating nuts helps prevent = the=20 development of
diabetes and that zinc supplementation in obese,=20
insulin-resistant women improves insulin sensitivity, even in
the absence= of=20 zinc deficiency.

"Major constituents of nuts (unsaturated fatty acids,
magnesium and fiber= ) have=20 been inversely associated with
risk of type 2 diabetes; however, the over= all=20 effects of
nut consumption on risk of type 2 diabetes are not
available,"= write=20 Rui Jiang and colleagues from Brigham
and Women's Hospital in Boston. "Ou= r=20 results suggest that
frequent nut consumption is associated with lower ri= sk of=20
type 2 diabetes in women."

In this offshoot of the Nurses' Health Study, the authors
analyzed data f= rom=20 validated dietary questionnaires
completed in 1980 by 83,818 women, aged = 34 to=20 59
years, without a history of cardiovascular disease, cancer,
or diabete=
s.=20 During 16 years of follow-up, 3,206 women developed type
2 diabetes.

After adjustment for age, body mass index (BMI), smoking,
physical activi= ty,=20 family history of diabetes, alcohol,
and total energy intake, nut consump= tion=20 was inversely
associated with risk of diabetes. Compared with women who a=
lmost=20 never ate nuts, multivariate relative risk (RR)
among women who ate nuts = less=20 than once weekly was 0.93
(95% confidence interval [CI], 0.86-1.01). RR w= as 0.84=20
(95% CI, 0.76-0.93) among those who ate nuts one to four
times weekly, an= d RR=20 was 0.72 (95% CI, 0.60-0.88) among
those who ate nuts more than five time= s=20 weekly (P<.0001
for trend).

The protective effect of nuts did not vary after further
controlling for = dietary=20 factors, including intake of
vegetables, fruits, and whole grain, and a=20 composite diet
score composed of trans fat, cereal fiber, glycemic load, =
marine=20 omega-3 fatty acids, folate, and polyunsaturated
fat-to-saturated fat rat=
sa.

"The inverse association persisted within strata defined by
levels of fam= ily=20 history of diabetes, body mass index,
smoking, alcohol use, physical acti= vity,=20 and dietary
variables," the authors write.

In a separate study by Dilina N. Marreiro and colleagues from
Universidad= e de=20 Sao Paulo-SP in Brazil, zinc
supplementation enhanced insulin sensitivity= in=20 obese
women who were not zinc-deficient.

In this prospective, double-blind, clinical interventional
study, 56 obes= e women=20 with normal glucose tolerance were
randomized to treatment with zinc, 30 = mg=20 daily, or
placebo for four weeks. At baseline, age was 25 to 45 years,
me= an BMI=20 was 36.2 =B1 2.3 kg/m2, and both groups were
similar in clinical and labo= ratory=20 parameters.

Insulin resistance as measured by a homeostasis model
assessment did not = change=20 in the placebo group, but it
decreased from 5.8 =B1 2.6 to 4.3 =B1 1.7 (P= <.05) in=20 the
zinc-supplemented group. Insulin decreased from 28.8 =B1 14.1
to 21.2= =B1 8.1=20
sb/mL (P<.05) in the zinc group but was unchanged in the
placebo group.

"A short time of zinc supplementation improved insulin
sensitivity in obe= se=20 insulin [resistant] women without
zinc deficiency," the authors write. "A= lthough=20 the
mechanism concerning the effect of zinc supplementation is not
comple= tely=20 understood, further studies are recommended to
address the possible role = of zinc=20 therapy in insulin
resistance states such as [diabetes]."

ADA Annual Meeting: Abstracts 1644-P, 569-P. June 16-17, 2002.

--Tom Matthews

MoreLife for the rational - http://morelife.org Reality based
tools for More Life in quantity & quality

John 'The
Wed, Jun-26-02, 23:55
Once upon a time, our fellow Tom Matthews rambled on about
"Nuts and zinc (even in the absence of deficiency!) lower
NIDDM risk." Our champion De-Medicalizing in sci.med.nutrition
retorts, thusly ...

>Medscape Medical News Nuts May Lower Risk of Diabetes

>Laurie Barclay, MD

>Medscape Medical News 2002. © 2002 Medscape

>June 19, 2002 -- New data presented June 16-17 at the 62nd
>Scientific Sessions of the American Diabetes Association
>suggest that eating nuts helps prevent the development of
>diabetes and that zinc supplementation in obese,
>insulin-resistant women improves insulin sensitivity, even in
>the absence of zinc deficiency.

In my Health-with-Attitude mailing list, I add comments to
the latest research findings. Nor, do I violate copyrights
either. :-(
--
John Gohde, Achieving good Health is an Art, NOT a Science!

Health-with-Attitude is a support group for people trying to
follow a Healthy Lifestyle.
http://groups.yahoo.com/group/Health-with-Attitude/

Alex Brand
Sat, Jun-29-02, 20:56
On Wed, 26 Jun 2002, Tom Matthews wrote:
> Medscape Medical News Nuts May Lower Risk of Diabetes
>
> Laurie Barclay, MD
>
> Medscape Medical News 2002. =A9 2002 Medscape
>
> June 19, 2002 -- New data presented June 16-17 at the 62nd
> Scientific Ses=
sions of
> the American Diabetes Association suggest that eating nuts
> helps prevent =
the
> development of diabetes and that zinc supplementation in
> obese, insulin-resistant women improves insulin sensitivity,
> even in the absence=
of
> zinc deficiency.
>
> In a separate study by Dilina N. Marreiro and colleagues
> from Universidad=
e de
> Sao Paulo-SP in Brazil, zinc supplementation enhanced
> insulin sensitivity=
in
> obese women who were not zinc-deficient.
>
> In this prospective, double-blind, clinical interventional
> study, 56 obes=
e women
> with normal glucose tolerance were randomized to treatment
> with zinc, 30 =
mg
> daily, or placebo for four weeks. At baseline, age was 25 to
> 45 years, me=
an BMI
> was 36.2 =B1 2.3 kg/m2, and both groups were similar in
> clinical and labo=
ratory
> parameters.
>
> Insulin resistance as measured by a homeostasis model
> assessment did not =
change
> in the placebo group, but it decreased from 5.8 =B1 2.6 to
> 4.3 =B1 1.7 (P=
<.05) in
> the zinc-supplemented group. Insulin decreased from 28.8 =B1
> 14.1 to 21.2=
=B1 8.1
> mU/mL (P<.05) in the zinc group but was unchanged in the
> placebo group.
>
> "A short time of zinc supplementation improved insulin
> sensitivity in obe=
se
> insulin [resistant] women without zinc deficiency," the
> authors write. "A=
lthough
> the mechanism concerning the effect of zinc supplementation
> is not comple=
tely
> understood, further studies are recommended to address the
> possible role =
of zinc
> therapy in insulin resistance states such as [diabetes]."

This makes me wonder how they assessed and defined "zinc
deficiency".

Tom Matthe
Sun, Jun-30-02, 06:55
Alex Brands wrote:

> On Wed, 26 Jun 2002, Tom Matthews wrote:
>=20
>>Medscape Medical News Nuts May Lower Risk of Diabetes
>>
>>Laurie Barclay, MD
>>
>>Medscape Medical News 2002. =A9 2002 Medscape
>>
>>June 19, 2002 -- New data presented June 16-17 at the 62nd
>>Scientific S=
essions of
>>the American Diabetes Association suggest that eating nuts
>>helps preven=
t the
>>development of diabetes and that zinc supplementation in
>>obese, insulin-resistant women improves insulin sensitivity,
>>even in the absen=
ce of
>>zinc deficiency.
>>
>>In a separate study by Dilina N. Marreiro and colleagues
>>from Universid=
ade de
>>Sao Paulo-SP in Brazil, zinc supplementation enhanced
>>insulin sensitivi=
ty in
>>obese women who were not zinc-deficient.
>>
>>In this prospective, double-blind, clinical interventional
>>study, 56 ob=
ese women
>>with normal glucose tolerance were randomized to treatment
>>with zinc, 3=
mg
>>daily, or placebo for four weeks. At baseline, age was 25 to
>>45 years, =
mean BMI
>>was 36.2 =B1 2.3 kg/m2, and both groups were similar in
>>clinical and la=
boratory
>>parameters.
>>
>>Insulin resistance as measured by a homeostasis model
>>assessment did no=
t change
>>in the placebo group, but it decreased from 5.8 =B1 2.6 to
>>4.3 =B1 1.7 =
(P<.05) in
>>the zinc-supplemented group. Insulin decreased from 28.8 =B1
>>14.1 to 21=
=2E2 =B1 8.1
>>mU/mL (P<.05) in the zinc group but was unchanged in the
>> placebo group.=

>>
>>"A short time of zinc supplementation improved insulin
>>sensitivity in o=
bese
>>insulin [resistant] women without zinc deficiency," the
>>authors write. =
"Although
>>the mechanism concerning the effect of zinc supplementation
>>is not comp=
letely
>>understood, further studies are recommended to address the
>>possible rol=
e of zinc
>>therapy in insulin resistance states such as [diabetes]."
>>
>=20
> This makes me wonder how they assessed and defined "zinc
> deficiency".

I assume that they used the standard definition which is
essentially: "Zinc is deficient if the patient shows the signs
which cause zinc to be =

classified as an essential mineral". Perhaps they both found
no symptoms = and=20 also measured blood levels as within the
normal (non-deficient) range.

What the study really shows, of course, is that the term
"deficient" as n= ormally=20 defined in nutrition is quite
arbitrary and insufficient. "Not deficient"= does=20 not mean
sufficient to prevent all possible types of negative symptoms
wh= ich=20 that nutrient could prevent. The definitions of
both "deficiency" and "ad= equacy"=20 are also problematic
because of individual variation. Indeed, it is even =

possible that what is a deficient level of some nutrient for
one person c= ould be=20 a toxic level for another.

--Tom Matthews

MoreLife for the rational - http://morelife.org Reality based
tools for More Life in quantity & quality