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Watchman
Sun, Apr-28-02, 01:01
I am hoping someone might take the time to answer this
question ..

For that little kid over there ..

In studying I ran across this snip from a IV feeding site and
came across what I thought to be an interesting point.

I believe it states .. high lipids lead to inflammatory
condition ..?

Because of this low rate of blood flow, the venous
epithelium of the superficial veins is highly susceptible
to injury from hyperosmolar infusions. The net result is
local inflammation that further reduces blood flow and
accentuates the injury response, inducing phlebitis.

For example, the clinical course of adult respiratory
distress syndrome (ARDS) can worsen with the
administration of large quantities of lipid emulsion.(14)
For these reasons, acutely ill patients with moderate to
severe metabolic stress usually cannot be fed entirely by
PPN therapy.

Subject: rate of blood flow/microvessels

Because of the relatively low rates of blood flow through
the peripheral veins, the ability of smaller blood vessels
to withstand concentrated parenteral nutrition admixtures
is severely limited. Hemodilution plays an important role
in the lifespan of indwelling intravascular catheters
principally, but not exclusively, by reducing osmotic
injury to the vein intima. Unlike the large central veins
used for TPN therapy, where blood flow approaches 2500
mL/min, flow through the superficial veins is approximately
25 to 50 mL/min.

Because of this low rate of blood flow, the venous
epithelium of the superficial veins is highly susceptible
to injury from hyperosmolar infusions. The net result is
local inflammation that further reduces blood flow and
accentuates the injury response, inducing phlebitis. The
osmolarity of the final admixture is a major factor in the
development of peripheral vein phlebitis. To reduce the
incidence of phlebitis from osmotic injury, PPN admixtures
are formulated so that the final osmolarity is maintained
at or below 900 mOsm/L.

Concentrations above this level dramatically increase the
risk of phlebitis.(9,10) When the concentration exceeds
900mOsm/L, the ability of the peripheral veins to dilute
parenteral infusions sufficiently is compromised, and
chemical irritation of the vein intima occurs. Admixtures
greater than 600 to 900 mOsm/L are associated with
increased rates of phlebitis.(9,10) Likewise,
concentrations of potassium chloride exceeding 40 mEq/L(10)
and acidic infusions(11,12) are additional risk factors for
phlebitis. Finally, for similar reasons, patients with
circulatory abnormalities (e.g., diabetes mellitus) may
have poor venous access or may not tolerate peripheral vein
infusions. Candidates for PPN therapy should be evaluated
by an experienced clinician to ensure that sufficient
peripheral vein access is available for the projected
course of therapy.

Clinical Considerations

Because of the limited ability of the peripheral veins to
tolerate concentrated nutritional admixtures, large volumes
of fluid are necessary if the patient's nutritional needs
are to be met solely by PPN therapy. Table 8-1 identifies
diluted PPN admixtures that maintain an osmolarity at 900
mOsm/L or less. Although this level may be achieved in
patients with good cardiopulmonary and renal function, it
may not be possible in many hospitalized patients with
limited organ function. Volume overload is common in the
critical care setting and may complicate the management of
these patients.(13) Similarly, disorders that cause fluid
retention (e.g., congestive heart failure, liver disease,
renal failure) may pose additional clinical concerns. For
these reasons, PPN therapy is not indicated as the primary
source of nutrition support in these patients.

Lipid emulsions are a dense caloric source. They are
essentially isosmotic with blood and can be substituted for
a portion of the glucose calories to permit the patient's
total energy needs to be met with PPN therapy. Lipid
emulsions can reduce the fluid burden in these patients
when these components account for 60% or more of the total
caloric intake. Again, the management of certain clinical
conditions
(i.e., hyperlipidemias, particularly hypertriglyceridemia;
sepsis; liver disease) may be affected by high
concentrations of exogenous lipids. Moreover, because
linoleic acid is a precursor to the vasoactive
prostaglandins, additional complications may ensue.
For example, the clinical course of adult respiratory
distress syndrome (ARDS) can worsen with the
administration of large quantities of lipid
emulsion.(14) For these reasons, acutely ill patients
with moderate to severe metabolic stress usually
cannot be fed entirely by PPN therapy.

--
Jesus was a Vegetarian! http://www.nucleus.com/watchman Moses
was a Mystic! http://www.nucleus.com/watchman/light.html

Aimee G
Sun, Apr-28-02, 01:01
"watchman" <watchman@nucleus.com> wrote in message
news:3cc03056@news.nucleus.com...
> I am hoping someone might take the time to answer this
> question ..
>
> For that little kid over there ..
>
> In studying I ran across this snip from a IV feeding site
> and came across what I thought to be an interesting point.
>
> I believe it states .. high lipids lead to inflammatory
> condition ..?
>
>
> Because of this low rate of blood flow, the venous
> epithelium of the superficial veins is highly susceptible
> to injury from hyperosmolar infusions. The net result is
> local inflammation that further reduces blood flow and
> accentuates the injury response, inducing phlebitis.
>

This paragraph is important. Peripheral vessels cannot handle
heavy infusion loads. Central lines are less susceptibe to the
damage. Lipids are pretty big molecules; envision this, take a
garden hose and a 4 inch pipe. Drop some gravel and water down
them. Which do you think would be more "irritated"? It's not
the lipids persay, but the size of the traffic on the road.

Slenon
Sun, Apr-28-02, 01:01
Watchman wonders in the wilderness of his own mind:
> For that little kid over there .. > In studying I ran across
> this snip
from a IV feeding site and came across > what I thought to be
an interesting
>point. > I believe it states .. high lipids lead to
>inflammatory condition
..?

And the point of your post, Tommy, would be something
beyond taking your safety scissors and adding wasted
bandwidth to three usegroups? You, who have publicly
screamed your hatred of science, biology, and all people
medical, lack the basic education to assimilate the
articles you spend your days posting to multiple usegroups.
Every message you post adds to the number of people
world-wide who find you delusional. Get help.

--
Stev Still dancing in the Phil Zone & scattering Garcia ashes
Stev Lenon MT(ASCP) - In healthcare the ultimate bottom line
is patients not profit Save a cow, eat a PETA member
slenon@tampabay.rr.com http://web.tampabay.rr.com/stevglo/ind-
ex.html/slhomepage92kword.htm

Watchman
Sun, Apr-28-02, 01:01
In sci.med.laboratory slenon <slenon@tampabay.rr.com> wrote:
> Watchman wonders in the wilderness of his own mind:
>> For that little kid over there .. > In studying I ran
>> across this snip
> from a IV feeding site and came across > what I thought to
> be an interesting
>>point. > I believe it states .. high lipids lead to
>>inflammatory condition
> ..?

> And the point of your post, Tommy, would be something beyond
> taking your safety scissors and adding wasted bandwidth to
> three usegroups?

If you don't know Steve .. crossposting saves bandwith ..
rather than posting to different newsgroups ..

Contrary to what I've seen in YOU .. Steve .. I believe there
may be a few people here who .. 'know their stuff' .. and are
actually 'in it' because the give a damn .. about that 'little
kid over there' ..

> Every message you post adds to the number of people
> world-wide who find you delusional. Get help.

Only in those people who are biased .. pal ..

Hyperviscosity .. one of the causes is polycythemia .. too
many red blood cells .. and CONTRARY to what you say ..
excess iron DOES cause polycythemia .. as evidenced in
poisoning by iron.

To those who are interested ..

I have been studying the effects of hyperviscosity / thick
blood / erytrocytosis .. too many red blood cells in the body.
This is due to what I believe to be the effects of too much
iron. I believe as in the treatment of anemia when iron is
used to raise red blood cells .. too much iron would raise TOO
MANY red blood cells .

This would lead to thick blood and therefore lack of
circulation through the microvessels in the body .. milkshake
versus water.

I have been looking into gastrointestinal disorders.

Trauma is KNOWN to induce hyperviscosity / thick blood .. and
trauma is KNOWN to induce 'colitis'.

I believe this to be the lack of circulation through the
microvessels that line the gut .. as evidenced I believe in
the high rate of necrotizing enteritis in the newborn who
present with polycythemia / thick blood / too many red
blood cells.

Restriction of a rat .. leads to hyperviscosity .. stress .

I find the below to be interesting ..

Ischemia - low blood flow ..

Who loves ya. Tom Jesus was a Vegetarian!
http://www.nucleus.com/watchman Moses was a Mystic!
http://www.nucleus.com/watchman/light.html

J Physiol Pharmacol 2002 Mar;53(1):39-50

Effects of reactive oxygen species action on gastric mucosa in
various models of mucosal injury.

Kwiecien S, Brzozowski T, Konturek SJ

Department of Physiology, Jagiellonian University School of
Medicine, Cracow, Poland.

[Medline record in process]

BACKGROUND: The exposure of gastric mucosa to damaging
factors, such as ethanol, water restraint stress, or
ischemia followed by reperfusion, produces pathological
changes: inflammatory process, hemorrhagic erosions, even
acute ulcers. The base of these changes is a disturbance of
protective mechanisms and disrupture of gastric mucosal
barrier. Previous studies pointed out the role of
disturbances of gastric blood flow, mucus secretion and
involvement of prostaglandins and nitric oxide formation in
the pathomechanism of gastric mucosa lesions. The role of
reactive oxygen species (ROS) in these processes has been
little studied. Aim: The purpose of our present
investigations is to explain the participation of ROS in
acute gastric mucosal damage by various irritants. MATERIAL
AND METHODS: Experiments were carrying out on 80 male
Wistar rats. To assess gastric blood flow (GBF) laser
Doppler flowmeter was used. The area of gastric lesions was
established by planimetry. The levels of proinflammatory
cytokines were measured by ELISA technique. The
colorimetric assays were used to determine of
malondialdehyde (MDA) and 4-hydroxynonenal (4-HNE) as well
as superoxide dismutase (SOD) activity. RESULTS: We
demonstrated that 3.5 h of water immersion and restraint
stress (WRS), 30 min of gastric ischemia followed by 60 min
of reperfusion or intragastric administration of 100%
ethanol, all resulted in appearance of acute gastric
mucosal lesions accompanied by a significant decrease of
gastric blood flow. These lesions are also accompanied by
the significant increase of proinflammatory cytokines
including interleukin-1 beta (IL-1beta) and tumor necrosis
factor alpha (TNFalpha) plasma level. Biological effects of
ROS were estimated by measuring tissue level of MDA and
4-HNE, the products of lipid peroxydation by ROS, as well
as the activity of SOD, the scavanger of ROS. It was
established that 3.5 h of WRS, ischemia-reperfusion and
100% ethanol lead to significant increase of MDA and 4-HNE
mucosal level, accompanied by a decrease of SOD activity
(significant in WRS and ethanol application). CONCLUSIONS:
The pathogenesis of experimental mucosal damage in rat
stomach includes the generation of ROS that seem to play an
important role, namely due to generation of lipid
peroxides, accompanied by impairment of antioxidative
enzyme activity of cells.

PMID: 11939718, UI: 21936776
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Who loves ya. Tom
--
Jesus was a Vegetarian! http://www.nucleus.com/watchman Moses
was a Mystic! http://www.nucleus.com/watchman/light.html

Slenon
Sun, Apr-28-02, 01:01
Watchman tirade snipped

Buzz off Tommy, cross posting is your way of seeking attention
and support.

You've been studying gastrointestinal disorders? Well, I've
suppose you've inserted your head far enough to have a good
view of the inner workings of your GI tract.

You have a marvelous ability to take two words and string them
together to form a conclusion with absolutely no foundation
save that which exists in the wasteland of your own mind.

You are dangerous to yourself and potentially to others. Get
mental health help now.

--
Stev Still dancing in the Phil Zone & scattering Garcia ashes
Stev Lenon MT(ASCP) - In healthcare the ultimate bottom line
is patients not profit Save a cow, eat a PETA member
slenon@tampabay.rr.com http://web.tampabay.rr.com/stevglo/ind-
ex.html/slhomepage92kword.htm