PDA

View Full Version : Iron accumulation / chronic hepatitis C


Welcome to the Active Low-Carber Forums

Support for Atkins diet, Protein Power, Neanderthin (Paleo Diet), CAD/CALP, Dr. Bernstein Diabetes Solution and any other healthy low-carb diet or plan, all are welcome in our lowcarb community. Forget starvation and fad diets -- join the healthy eating crowd! You may register by clicking here, it's free!



Ironjustic
Sun, Jan-22-06, 17:16
<<snip>> Severe fibrosis and cirrhosis were associated more
markedly with the presence of hemosiderin iron <<snip>>

Am J Clin Pathol. 2005 Dec;124(6):846-53. Related
Articles, Links

Iron accumulation in chronic hepatitis C: relation of hepatic
iron distribution, HFE genotype, and disease course.

Corengia C, Galimberti S, Bovo G, Vergani A, Arosio C, Mariani
R, Redaelli A, Riva A, Cestari C, Pozzi M, Valsecchi MG,
Piperno A.

Unit of Clinical Medicine, Department of Clinical Medicine,
Prevention and Biotechnologies, University of Milano-Bicocca,
San Gerardo Hospital, Monza, Italy.

The aim of the present study was to describe the
histopathologic features of hepatic iron accumulation in
patients with chronic hepatitis C (CH-C) infection, the
relation between HFE mutations and hepatic iron location and
among iron distribution, HFE, and hepatic damage. We studied
206 patients with CH-C infection. Of 101 patients with
hemosiderin deposits, 90.1% had iron deposits in hepatocytes
(alone or with sinusoidal and/or portal involvement). The
hepatic iron score increased significantly as iron
accumulation involved sinusoidal and portal tract compartments
and according to HFE genotypes. Severe fibrosis and cirrhosis
were associated more markedly with the presence of hemosiderin
iron in the 3 hepatic compartments, HFE mutations, and high
alcohol intake. We suggest that part of the iron accumulation
in CH-C infection derives from increased iron absorption and
release from storage cells and that the amount and
distribution of hepatic iron deposits is related to hepatic
damage. HFE mutations favor both processes, but other factors,
genetic or acquired, are involved.

PMID: 16416733 [PubMed - in process]

--------------------------------------------------------------
-------------------
Who loves ya. Tom

Jesus Was A Vegetarian! http://jesuswasavegetarian.7h.com

Man Is A Herbivore!
http://pages.ivillage.com/ironjustice/manisaherbivore

DEAD PEOPLE WALKING
http://pages.ivillage.com/ironjustice/deadpeoplewalking

Gib
Sun, Jan-22-06, 17:16
<ironjustice@aol.com> wrote in message
news:1137930529.798080.101350@g44g2000cwa.googlegroups.com...
> <<snip>> Severe fibrosis and cirrhosis were associated more
> markedly with the presence of hemosiderin iron <<snip>>
>
> Am J Clin Pathol. 2005 Dec;124(6):846-53. Related
> Articles, Links
>
>
> Iron accumulation in chronic hepatitis C: relation of
> hepatic iron distribution, HFE genotype, and disease course.
>
> Corengia C, Galimberti S, Bovo G, Vergani A, Arosio C,
> Mariani R, Redaelli A, Riva A, Cestari C, Pozzi M, Valsecchi
> MG, Piperno A.
>
> Unit of Clinical Medicine, Department of Clinical Medicine,
> Prevention and Biotechnologies, University of
> Milano-Bicocca, San Gerardo Hospital, Monza, Italy.
>
> The aim of the present study was to describe the
> histopathologic features of hepatic iron accumulation in
> patients with chronic hepatitis C (CH-C) infection, the
> relation between HFE mutations and hepatic iron location and
> among iron distribution, HFE, and hepatic damage. We studied
> 206 patients with CH-C infection. Of 101 patients with
> hemosiderin deposits, 90.1% had iron deposits in hepatocytes
> (alone or with sinusoidal and/or portal involvement). The
> hepatic iron score increased significantly as iron
> accumulation involved sinusoidal and portal tract
> compartments and according to HFE genotypes. Severe fibrosis
> and cirrhosis were associated more markedly with the
> presence of hemosiderin iron in the 3 hepatic compartments,
> HFE mutations, and high alcohol intake. We suggest that part
> of the iron accumulation in CH-C infection derives from
> increased iron absorption and release from storage cells and
> that the amount and distribution of hepatic iron deposits is
> related to hepatic damage. HFE mutations favor both
> processes, but other factors, genetic or acquired, are
> involved.
>
> PMID: 16416733 [PubMed - in process]
>
> ------------------------------------------------------------
> ---------------
------
> Who loves ya. Tom
>
>
> Jesus Was A Vegetarian! http://jesuswasavegetarian.7h.com
>
>
> Man Is A Herbivore!
> http://pages.ivillage.com/ironjustice/manisaherbivore
>
>
> DEAD PEOPLE WALKING
> http://pages.ivillage.com/ironjustice/deadpeoplewalking

Perhaps this could help. My doctor prescribed this med but
before I received it periodic phlebotomies brought my Ferritin
level from a high of 6000 during tx to 33 post tx. I also
suffer from a genetic disorder, hemochromatosis , that causes
iron overload.

http://www.exjade.com/index.jsp
----------------------------
http://www.drugs.com/nda/exjade_050622.html
----------------------------
http://www.medicalnewstoday.com/medicalnews.php?newsid=23895